Posted by iforgotmypassword on April 27, 2008, at 16:15:43 [reposted on April 27, 2008, at 16:24:25 | original URL]
Of course, I mean in relation to the method of doing so via inhibition of MAO-B in the brain.
Abstracts briefly endorse safety, and I have no access to possible elaboration that there may be in full-texts, however has ANYONE come across anything that would fully explain that chronic unmoderated PEA activity in the brain is, or must logically be: **completely safe.** (?)
Any reasoning behind simply regarding it as safe? SSRIs prevent the moderation of serotonin, which as we have discovered can be harmful. Increasing PEA through exogenous sources and inhibiting it's moderation may not, in future, prove to be a similar case? After all, our documented experience with this combination is sparse, and it took us forever to articulate that YES, SSRIs are hurting people.
This, obviously, but for clarification is about the selegiline DLPA or similar brain PEA level increasing and preserving combinations.
I am willing to try anything at this point, but that's what made me turn to Paxil years upon years ago and I still regret it, and it didn't even help me. (However selegiline DLPA might. **Hard to balance.**)
poster:iforgotmypassword
thread:825869
URL: http://www.dr-bob.org/babble/neuro/20080418/msgs/825869.html