Posted by rca on December 29, 2012, at 9:29:19
In reply to Re: Bifidobacteria reduce depression » rca, posted by larryhoover on December 28, 2012, at 10:44:25
Thanks for your thoughtful reply - haven't "digested" it all but can respond to one comment here.
>High-fat HIGH-CARB combination. There it is, plain as day.
The study (quote below) involved comparison of a high fat high carb (HFHC) group compared to an American Heart Association recommended food group (AHA). But you know what they were actually eating? The HFHC groups ate egg muffin and sausage muffin sandwiches and two hash browns, which contain 88 g carbohydrates, 51 g fat [33% saturated] and 34 g protein [carbohydrates 41%, protein 17%, and fat 42%]; the second group ate oatmeal, milk, orange juice, raisins, peanut butter, and English muffin (carbohydrates 58%, protein 15%, and fat 27%). I think their choice of these two groups were unfortunate and their labeling misleading.
The HFHC group ate a typical SAD diet of animal products and simple carbohydrates; the second group still included some animal product and was predominately a high carbohydrate meal, but had a significant amount of complex carbohydrates too.
Their data indicated a pronounced inflammation after the HFHC (SAD) diet compared to the AHA diet (even with the milk , juice, and English muffin Id prefer comparing to a whole plant based diet something like whole rolled oats, blueberries, crushed flax seeds in soy milk).
The entire article is available on-line here: http://www.ncbi.nlm.nih.gov/pubmed?term=Diabetes%20Care.%202009%20Dec%3B%2032(12)%3A2281-7.
Heres the interesting part. Please examine the two figures (each with three figures on them), showing the inflammatory marker rise (figure 1) and the endotoxin rise (Figure 2). Notice anything strange? In all cases these parameters rise in just ONE HOUR after the meal certainly not time enough to reach the leaky gut colon where all the bacteria are. I see no explanation other than there are endotoxins in the animal products that are being absorbed in the small intestine.
Yep. Look at (Erridge, 2011) below. For the first time ever, they aimed to determine whether common foodstuffs may contain appreciable quantities of endotoxin. Forty extracts were therefore prepared from twenty-seven foodstuffs common to the Western diet, and the capacity of each to induce the secretion of inflammatory signals from human white blood cells was measured. They found whopping doses of endotoxin equivalents in pork, poultry, dairy.
Conclusion is that animal products may cause inflammation because of their endotoxins.
Ghanim H, Abuaysheh S, Sia CL, Korzeniewski K, Chaudhuri A, Fernandez-Real JM, Dandona P. Increase in plasma endotoxin concentrations and the expression of Toll-like receptors and suppressor of cytokine signaling-3 in mononuclear cells after a high-fat, high-carbohydrate meal: implications for insulin resistance. Diabetes Care. 2009 Dec; 32(12):2281-7.Erridge C. The capacity of foodstuffs to induce innate immune activation of human monocytes in vitro is dependent on food content of stimulants of Toll-like receptors 2 and 4. Br J Nutr. 2011 Jan; 105(1):15-23. http://www.ncbi.nlm.nih.gov/pubmed/20849668
poster:rca
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URL: http://www.dr-bob.org/babble/20121217/msgs/1034122.html